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Dermatology3 papers

Toxic acute tubular necrosis

Last edited: 4/15/2026

Overview

Toxic acute tubular necrosis (TAN) is a severe form of acute kidney injury characterized by widespread necrosis of renal tubular cells, often secondary to severe systemic insults such as sepsis, shock, or certain medications like nephrotoxins.

Diagnosis

  • Clinical Presentation: Acute kidney injury with oliguria or anuria, elevated serum creatinine, and characteristic changes on urinalysis (hematuria, cylindruria, granular casts) 1.
  • Laboratory Tests: Elevated serum creatinine, blood urea nitrogen (BUN), and fractional excretion of sodium (FENa) <1% 1.
  • Imaging: Renal ultrasound may show reduced kidney size or increased echogenicity 1.
  • Renal Biopsy: Definitive diagnosis showing tubular cell necrosis and inflammation (indicated in refractory cases) 1.
  • Management

  • Fluid Management: Aggressive fluid resuscitation to maintain hemodynamic stability 1.
  • Control Underlying Cause: Prompt discontinuation of nephrotoxic agents and management of underlying conditions (e.g., sepsis, shock) 1.
  • Supportive Care: Maintenance of adequate nutrition, electrolyte balance, and acid-base status 1.
  • Dialysis: Initiation of renal replacement therapy (RRT) in cases of severe hyperkalemia, acidosis, or fluid overload 1.
  • Monitoring: Frequent monitoring of renal function, electrolytes, and fluid balance 1.
  • Special Populations

  • Pregnancy: Management focuses on maternal stability and fetal monitoring; RRT may be necessary with careful consideration of risks 1.
  • Pediatrics: Tailored fluid and electrolyte management; close monitoring for developmental impacts 1.
  • Elderly: Emphasis on minimizing complications and optimizing supportive care due to increased comorbidities 1.
  • Comorbidities: Careful management of coexisting conditions to prevent exacerbation; individualized treatment plans 1.
  • Key Recommendations

  • Early Recognition and Management of Underlying Causes: Prompt identification and treatment of precipitating factors such as sepsis and nephrotoxic drug exposure (Evidence: Strong) 1.
  • Aggressive Fluid Resuscitation: Initiate early and maintain adequate hydration to support hemodynamic stability (Evidence: Strong) 1.
  • Initiate Renal Replacement Therapy When Indicated: Use RRT for severe metabolic disturbances or fluid overload (Evidence: Moderate) 1.
  • Comprehensive Monitoring: Regular assessment of renal function, electrolytes, and fluid status to guide treatment adjustments (Evidence: Moderate) 1.
  • Specialized Care for Vulnerable Populations: Tailor management strategies for pregnant women, children, and elderly patients considering their unique needs (Evidence: Expert opinion) 1.
  • References

    1 Tasker F, Smith SP, Mohd Mustapa MF, de Berker DAR. British Association of Dermatologists national clinical audit on the management of Stevens-Johnson syndrome/toxic epidermal necrolysis in adults. Clinical and experimental dermatology 2024. link

    Original source

    1. [1]
      British Association of Dermatologists national clinical audit on the management of Stevens-Johnson syndrome/toxic epidermal necrolysis in adults.Tasker F, Smith SP, Mohd Mustapa MF, de Berker DAR Clinical and experimental dermatology (2024)

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