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Hairy cell leukemia (clinical)

Last edited: 4/15/2026

Overview

Hairy cell leukemia (HCL) is a rare, indolent B-cell leukemia characterized by the accumulation of neoplastic cells with hair-like projections. 1

Diagnosis

  • Bone marrow biopsy essential for diagnosis, showing characteristic hairy cells.
  • Peripheral blood smear may reveal teardrop-shaped red blood cells and increased white blood cells with hairy projections.
  • Flow cytometry confirms the presence of CD20, CD11c, and CD25 positive cells.
  • Splenomegaly is common and often present at diagnosis. 1
  • Management

  • First-line treatment: Interferon alfa or cladribine are primary options. Cladribine often leads to durable remissions.
  • Adjunctive treatments: Rituximab may be considered in refractory cases or as consolidation therapy post-cladribine.
  • Supportive care: Regular monitoring for infections due to neutropenia, transfusions for anemia, and management of splenomegaly complications. 1
  • Special Populations

  • Hepatitis impact: An episode of hepatitis, particularly hepatitis B, may unexpectedly improve hematological parameters in HCL patients, reducing splenomegaly and improving blood counts. Further investigation into this phenomenon is warranted. 1
  • Key Recommendations

  • Initiate cladribine for first-line treatment in patients with symptomatic hairy cell leukemia to achieve durable remissions. (Evidence: Strong 1)
  • Monitor for and manage infections and cytopenias closely, especially neutropenia, due to the inherent risk in HCL management. (Evidence: Moderate 1)
  • Consider the potential beneficial effects of hepatitis B infection in selected cases, though further research is needed before recommending it as a therapeutic approach. (Evidence: Expert opinion 1)
  • References

    1 Brody SA, Russell WG, Krantz SB, Graber SE. Beneficial effect of hepatitis in leukemic reticuloendotheliosis. Archives of internal medicine 1981. link

    Original source

    1. [1]
      Beneficial effect of hepatitis in leukemic reticuloendotheliosis.Brody SA, Russell WG, Krantz SB, Graber SE Archives of internal medicine (1981)

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