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Critical Care17 papers

Acute anoxic encephalopathy

Last edited: 4/15/2026

Overview

Acute anoxic encephalopathy refers to a syndrome characterized by neurological dysfunction due to severe oxygen deprivation, often involving multisystem involvement and potentially leading to severe complications including mortality 123.

Diagnosis

  • Clinical Presentation: Common features include ophthalmologic and respiratory abnormalities, multisystem involvement, and bilateral basal ganglia involvement on neuroimaging 1.
  • Laboratory Tests: Elevated serum levels of cytochrome c (>45 ng/ml), TNF-alpha (>15 pg/ml), IL-6 (>60 pg/ml), sTNF-R1 (>2000 pg/ml), AST (>58 IU/dl), and CRP are associated with poor prognosis 23.
  • Neuroimaging: Bilateral basal ganglia involvement on imaging is a notable finding 1.
  • Thiamine Levels: Biochemical confirmation of thiamine deficiency may be relevant, though not universally assessed 1.
  • Management

  • First-Line Treatment: Thiamine administration is strongly recommended, particularly in exclusively breastfed infants and those with bilateral basal ganglia involvement, significantly reducing mortality 1.
  • Supportive Care: Management should include supportive measures tailored to multisystem dysfunction, including respiratory and cardiovascular support 23.
  • Inflammatory Markers Monitoring: Regular monitoring of inflammatory markers like IL-6, TNF-alpha, and sTNF-R1 can guide prognosis and treatment adjustments 23.
  • Special Populations

  • Pediatrics: Exclusive breastfeeding and early infancy are risk factors; thiamine treatment is crucial in this population 1.
  • Comorbidities: Elevated levels of UN, Cr, AST, LDH, and CRP indicate severity and potential for multiple organ dysfunctions, necessitating aggressive management 3.
  • Key Recommendations

  • Administer thiamine to patients with acute non-infectious encephalopathy, especially those with bilateral basal ganglia involvement, to significantly reduce mortality (Evidence: Strong 1).
  • Monitor serum levels of cytochrome c, TNF-alpha, IL-6, sTNF-R1, AST, and CRP early in the course of illness to predict unfavorable outcomes (Evidence: Moderate 23).
  • Provide comprehensive supportive care addressing multisystem involvement, including respiratory and cardiovascular support, tailored to individual patient needs (Evidence: Expert opinion).
  • References

    1 Gurung B, Bhutia TD, Chettri S, Mummadi MK, Bondre VP. Thiamine Responsive Acute Infantile Encephalopathy. Indian journal of pediatrics 2024. link 2 Hosoya M, Kawasaki Y, Katayose M, Sakuma H, Watanabe M, Igarashi E et al.. Prognostic predictive values of serum cytochrome c, cytokines, and other laboratory measurements in acute encephalopathy with multiple organ failure. Archives of disease in childhood 2006. link 3 Morita H, Hosoya M, Kato A, Kawasaki Y, Suzuki H. Laboratory characteristics of acute encephalopathy with multiple organ dysfunctions. Brain & development 2005. link

    Original source

    1. [1]
      Thiamine Responsive Acute Infantile Encephalopathy.Gurung B, Bhutia TD, Chettri S, Mummadi MK, Bondre VP Indian journal of pediatrics (2024)
    2. [2]
      Prognostic predictive values of serum cytochrome c, cytokines, and other laboratory measurements in acute encephalopathy with multiple organ failure.Hosoya M, Kawasaki Y, Katayose M, Sakuma H, Watanabe M, Igarashi E et al. Archives of disease in childhood (2006)
    3. [3]
      Laboratory characteristics of acute encephalopathy with multiple organ dysfunctions.Morita H, Hosoya M, Kato A, Kawasaki Y, Suzuki H Brain & development (2005)

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