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Pediatrics24 papers

Anal atresia

Last edited: 4/14/2026

Overview

Anal atresia, also known as imperforate anus, is a congenital anomaly characterized by the absence or abnormal formation of the anal opening, often associated with other gastrointestinal tract malformations such as multiple atresias and imperforate anus 45.

Diagnosis

  • Clinical Presentation: Absence of anal opening, associated gastrointestinal atresias, and intraluminal calcifications on imaging 45.
  • Imaging: Abdominal radiographs showing intraluminal calcifications between areas of atresia 5.
  • Endoscopic Evaluation: To assess the extent of atresias and identify anatomical details 4.
  • Genetic Testing: Considered in cases with a family history or syndromic associations 3.
  • Management

  • Surgical Intervention: Primary repair or staged procedures depending on the complexity and associated anomalies 4.
  • Multidisciplinary Care: Collaboration with pediatric surgeons, gastroenterologists, and neonatologists 4.
  • Postoperative Care: Close monitoring for complications such as bowel obstruction and infection 4.
  • Nutritional Support: Early enteral feeding or parenteral nutrition as needed 4.
  • Special Populations

  • Pediatrics: Primary focus of management with high mortality rates reported in historical cases 45.
  • Comorbidities: Association with other congenital anomalies like hypoganglionosis, limb anomalies, and sensorineural deafness 2.
  • Key Recommendations

  • Early Surgical Intervention: Primary surgical repair should be considered promptly to prevent complications 4 (Evidence: Strong).
  • Multidisciplinary Approach: Essential for comprehensive care addressing both immediate and long-term outcomes 4 (Evidence: Strong).
  • Genetic Counseling: Recommended for families with a history of multiple gastrointestinal atresias, especially in populations with known genetic predispositions 5 (Evidence: Moderate).
  • References

    1 Zieba J, Forlenza KN, Khatra JS, Sarukhanov A, Duran I, Rigueur D et al.. TGFβ and BMP Dependent Cell Fate Changes Due to Loss of Filamin B Produces Disc Degeneration and Progressive Vertebral Fusions. PLoS genetics 2016. link 2 Kwok IH, Grant HW, Menon P. Trilogy of gastrointestinal atresias: a case report and review of the literature. Journal of pediatric surgery 2011. link 3 Fete M, vanBokhoven H, Clements SE, McKeon F, Roop DR, Koster MI et al.. International Research Symposium on Ankyloblepharon-Ectodermal Defects-Cleft Lip/Palate (AEC) syndrome. American journal of medical genetics. Part A 2009. link 4 Shen-Schwarz S, Fitko R. Multiple gastrointestinal atresias with imperforate anus: pathology and pathogenesis. American journal of medical genetics 1990. link 5 Daneman A, Martin DJ. A syndrome of multiple gastrointestinal atresias with intraluminal calcification. A report of a case and a review of the literature. Pediatric radiology 1979. link

    Original source

    1. [1]
      TGFβ and BMP Dependent Cell Fate Changes Due to Loss of Filamin B Produces Disc Degeneration and Progressive Vertebral Fusions.Zieba J, Forlenza KN, Khatra JS, Sarukhanov A, Duran I, Rigueur D et al. PLoS genetics (2016)
    2. [2]
      Trilogy of gastrointestinal atresias: a case report and review of the literature.Kwok IH, Grant HW, Menon P Journal of pediatric surgery (2011)
    3. [3]
      International Research Symposium on Ankyloblepharon-Ectodermal Defects-Cleft Lip/Palate (AEC) syndrome.Fete M, vanBokhoven H, Clements SE, McKeon F, Roop DR, Koster MI et al. American journal of medical genetics. Part A (2009)
    4. [4]
      Multiple gastrointestinal atresias with imperforate anus: pathology and pathogenesis.Shen-Schwarz S, Fitko R American journal of medical genetics (1990)
    5. [5]

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