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Cardiology17 papers

Exudative pleurisy

Last edited: 4/22/2026

Overview

Familial exudative vitreoretinopathy (FEVR) is a genetic disorder characterized by abnormal retinal angiogenesis leading to incomplete peripheral retinal vascularization, ischemia, and potential complications such as neovascularization, vitreous hemorrhage, and retinal detachment 135.

Diagnosis

  • Clinical Features: Temporal neovascularization, vitreous hemorrhage, retinal detachment 5.
  • Genetic Testing: Recommended for confirmation, focusing on genes like FZD4, LRP5, NDP, and TSPAN12 4.
  • Imaging: Multimodal imaging including fundus photography, fluorescein angiography, and OCT to assess retinal vascular abnormalities 1.
  • Chromosomal Analysis: Chromosomal microarray analysis to identify microduplications or deletions (e.g., 22q11.2) 3.
  • Management

  • Surgical Interventions: Active surgical management for complications like retinal detachment and vitreous hemorrhage 5.
  • Monitoring: Regular ophthalmological follow-up to monitor disease progression and manage complications 5.
  • Gene Therapy: Emerging potential, though specific protocols not detailed in current abstracts 4.
  • Special Populations

  • Pediatrics: Premature infants may present with overlapping symptoms of retinopathy of prematurity (ROP), necessitating careful differentiation 1.
  • Genetic Considerations: FEVR can be inherited in various patterns (autosomal dominant, recessive, X-linked), influencing genetic counseling and testing strategies 14.
  • Key Recommendations

  • Genetic Testing for Diagnosis: Perform genetic testing focusing on FZD4, LRP5, NDP, and TSPAN12 mutations for definitive diagnosis of FEVR (Evidence: Moderate) 4.
  • Regular Ophthalmological Monitoring: Schedule frequent ophthalmological evaluations to detect and manage complications early (Evidence: Expert opinion) 5.
  • Surgical Management for Complications: Employ surgical interventions for severe complications such as retinal detachment and vitreous hemorrhage (Evidence: Moderate) 5.
  • References

    1 López-Cañizares A, Lazzarini TA, Mendoza C, Berrocal AM. Familial exudative vitreoretinopathy (FEVR) in a child with novel microarray-defined deletion of 11q14 previously diagnosed as retinopathy of prematurity (ROP). Ophthalmic genetics 2023. link 2 Lu L, He K, Ni Y, Yu Z, Mao A. Exudative epidermitis of piglets caused by non-toxigenic Staphylococcus sciuri. Veterinary microbiology 2017. link 3 Gandhi JK, Tollefson TT, Telander DG. Falciform macular folds and chromosome 22q11.2: evidence in support of a locus for familial exudative vitreoretinopathy (FEVR). Ophthalmic genetics 2014. link 4 Robitaille JM, Zheng B, Wallace K, Beis MJ, Tatlidil C, Yang J et al.. The role of Frizzled-4 mutations in familial exudative vitreoretinopathy and Coats disease. The British journal of ophthalmology 2011. link 5 Bergen RL, Glassman R. Familial exudative vitreoretinopathy. Annals of ophthalmology 1983. link

    Original source

    1. [1]
    2. [2]
      Exudative epidermitis of piglets caused by non-toxigenic Staphylococcus sciuri.Lu L, He K, Ni Y, Yu Z, Mao A Veterinary microbiology (2017)
    3. [3]
    4. [4]
      The role of Frizzled-4 mutations in familial exudative vitreoretinopathy and Coats disease.Robitaille JM, Zheng B, Wallace K, Beis MJ, Tatlidil C, Yang J et al. The British journal of ophthalmology (2011)
    5. [5]
      Familial exudative vitreoretinopathy.Bergen RL, Glassman R Annals of ophthalmology (1983)

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