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Cardiology28 papers

Leigh's disease

Last edited: 4/15/2026

Overview

Leigh's disease, also known as subacute necrotizing encephalomyopathy, is a rare neurometabolic disorder characterized by progressive brain dysfunction due to mitochondrial dysfunction, leading to symptoms such as developmental regression, movement disorders, and respiratory issues 3.

Diagnosis

  • Clinical Presentation: Developmental regression, movement disorders, respiratory problems, and lactic acidosis 3.
  • Laboratory Tests: Elevated lactate and pyruvate levels in blood and cerebrospinal fluid 3.
  • Imaging: MRI often shows characteristic lesions in basal ganglia, brainstem, and cerebellum 3.
  • Genetic Testing: Identification of mutations in mitochondrial DNA or nuclear genes associated with mitochondrial function 3.
  • Management

  • Supportive Care: Management of symptoms including respiratory support, physical therapy, and nutritional support 3.
  • Dietary Modifications: Specific dietary adjustments may be considered based on metabolic profile, though specific recommendations are not detailed in the provided abstracts 3.
  • Medications: No specific drug treatments are highlighted in the abstracts provided 3.
  • Special Populations

  • Pediatrics: Leigh's disease predominantly affects infants and young children, with significant developmental impact 3.
  • Comorbidities: Cases may present with additional congenital anomalies, such as diaphragmatic hernia in syndromic associations, though not directly linked to Leigh's disease in the provided abstracts 1.
  • Key Recommendations

  • Early Identification and Monitoring: Regular monitoring of metabolic parameters (lactate, pyruvate) and neurological assessments to manage symptoms effectively (Evidence: Moderate 3).
  • Multidisciplinary Approach: Involvement of pediatric neurologists, metabolic specialists, and supportive care teams to address diverse clinical needs (Evidence: Expert opinion 3).
  • Genetic Counseling: Offer genetic counseling to families due to potential hereditary aspects, though specific inheritance patterns are not detailed in the abstracts (Evidence: Expert opinion 3).
  • References

    1 Addor MC, Stefanutti D, Farron F, Meinecke P, Lacombe D, Sarlangue J et al.. "C" trigonocephaly syndrome with diaphragmnatic hernia. Genetic counseling (Geneva, Switzerland) 1995. link 2 Nakagawa T, Maeshima M, Muto H, Kajiura H, Hattori H, Asahi T. Separation, amino-terminal sequence and cell-free synthesis of the smallest subunit of sweet potato cytochrome c oxidase. European journal of biochemistry 1987. link 3 Younossi-Hartenstein A, Vierbuchen M, Roth B, Schroeder R. Renal lesions in subacute encephalomyelopathy (Leigh's disease). The International journal of pediatric nephrology 1986. link

    Original source

    1. [1]
      "C" trigonocephaly syndrome with diaphragmnatic hernia.Addor MC, Stefanutti D, Farron F, Meinecke P, Lacombe D, Sarlangue J et al. Genetic counseling (Geneva, Switzerland) (1995)
    2. [2]
      Separation, amino-terminal sequence and cell-free synthesis of the smallest subunit of sweet potato cytochrome c oxidase.Nakagawa T, Maeshima M, Muto H, Kajiura H, Hattori H, Asahi T European journal of biochemistry (1987)
    3. [3]
      Renal lesions in subacute encephalomyelopathy (Leigh's disease).Younossi-Hartenstein A, Vierbuchen M, Roth B, Schroeder R The International journal of pediatric nephrology (1986)

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