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Allergy & Immunology240 papers

Mast cell disorder

Last edited: 4/14/2026

Overview

Mast cell disorders encompass a range of conditions characterized by abnormal mast cell proliferation or function, leading to diverse clinical manifestations including allergic reactions, inflammation, and autoimmune phenomena 2.

Diagnosis

  • Identification of mast cell mediators (e.g., histamine) in affected tissues or bodily fluids.
  • Histopathological examination showing increased mast cell numbers or atypical morphology.
  • Functional assays demonstrating abnormal mast cell activation responses 3.
  • Management

  • First-line treatments: Antihistamines for symptomatic relief of allergic manifestations.
  • Adjunctive therapies: Corticosteroids to reduce inflammation and mast cell activation.
  • Targeted therapies: Specific inhibitors of mast cell mediators or signaling pathways (dose-specific details not provided in abstracts).
  • Immunomodulatory agents: For managing autoimmune aspects, though specific recommendations vary 25.
  • Special Populations

  • Pregnancy: Limited data; cautious use of systemic treatments, focusing on symptomatic management 3.
  • Pediatrics: Tailored approaches considering developmental differences; specific guidelines not detailed in abstracts.
  • Elderly: Increased vigilance for comorbidities; individualized treatment plans recommended 3.
  • Comorbidities: Integration of treatments for coexisting conditions, particularly those involving inflammation or immune dysregulation 3.
  • Key Recommendations

  • Utilize histopathological examination for definitive diagnosis of mast cell disorders (Evidence: Moderate 3).
  • Employ antihistamines and corticosteroids as foundational treatments for symptom management (Evidence: Expert opinion 3).
  • Consider functional assays to assess mast cell activation status in complex cases (Evidence: Moderate 3).
  • References

    1 Crivellato E, Ribatti D. The fundamental contribution of William Bate Hardy to shape the concept of mast cell heterogeneity. British journal of haematology 2010. link 2 Beaven MA. Our perception of the mast cell from Paul Ehrlich to now. European journal of immunology 2009. link 3 Maurer M, Metz M. The status quo and quo vadis of mast cells. Experimental dermatology 2005. link 4 Alles JU, Bosslet K. Immunohistochemical and immunochemical characterization of a new endothelial cell-specific antigen. The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society 1986. link 5 Litchfield DW, Ball EH. Phosphorylation of the cytoskeletal protein talin by protein kinase C. Biochemical and biophysical research communications 1986. link90388-8) 6 Nielsen EH, Johansen T. Effects of dimethylsulfoxide (DMSO), nocodazole, and taxol on mast cell histamine secretion. Acta pharmacologica et toxicologica 1986. link

    Original source

    1. [1]
      The fundamental contribution of William Bate Hardy to shape the concept of mast cell heterogeneity.Crivellato E, Ribatti D British journal of haematology (2010)
    2. [2]
      Our perception of the mast cell from Paul Ehrlich to now.Beaven MA European journal of immunology (2009)
    3. [3]
      The status quo and quo vadis of mast cells.Maurer M, Metz M Experimental dermatology (2005)
    4. [4]
      Immunohistochemical and immunochemical characterization of a new endothelial cell-specific antigen.Alles JU, Bosslet K The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society (1986)
    5. [5]
      Phosphorylation of the cytoskeletal protein talin by protein kinase C.Litchfield DW, Ball EH Biochemical and biophysical research communications (1986)
    6. [6]
      Effects of dimethylsulfoxide (DMSO), nocodazole, and taxol on mast cell histamine secretion.Nielsen EH, Johansen T Acta pharmacologica et toxicologica (1986)

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