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Dilated cardiomyopathy

Last edited: 4/14/2026

Overview

Dilated cardiomyopathy (DCM) is a condition characterized by enlargement and weakened function of the heart's chambers, particularly the left ventricle, leading to reduced pumping efficiency and potential heart failure 4.

Diagnosis

  • Clinical Presentation: Symptoms include dyspnea, fatigue, and peripheral edema 4.
  • Physical Examination: May reveal signs of heart failure and murmurs 4.
  • Electrocardiogram (ECG): Often shows nonspecific changes but can indicate arrhythmias 4.
  • Echocardiography: Essential for confirming left ventricular dilation and reduced ejection fraction 4.
  • Cardiac MRI: Provides detailed assessment of myocardial structure and function 4.
  • Blood Tests: Include BNP levels and metabolic panel to assess heart failure severity 4.
  • Management

  • Medications:
  • - Angiotensin-Converting Enzyme Inhibitors (ACE inhibitors): Reduce mortality and hospitalization 4. - Angiotensin Receptor Blockers (ARBs): Alternative if ACE inhibitors are contraindicated 4. - Beta-Blockers: Improve survival and reduce hospitalizations 4. - Diuretics: Manage fluid overload and alleviate symptoms 4. - Aldosterone Antagonists: In severe cases, to reduce mortality 4.
  • Lifestyle Modifications: Dietary sodium restriction, fluid management, and exercise as tolerated 5.
  • Device Therapy: Implantable cardioverter-defibrillators (ICDs) for primary prevention in high-risk patients 4.
  • Heart Transplantation: Considered for end-stage refractory cases 4.
  • Special Populations

  • Pediatrics: Congenital syndromes like those involving cataracts and hypertrophic cardiomyopathy may coexist, necessitating multidisciplinary care 4.
  • Elderly: Management focuses on symptom relief and minimizing side effects due to polypharmacy concerns 5.
  • Comorbidities: Close monitoring and tailored treatment plans for coexisting conditions like hypertension or diabetes 4.
  • Key Recommendations

  • Use echocardiography for definitive diagnosis and monitoring of left ventricular function (Evidence: Strong 4).
  • Initiate ACE inhibitors or ARBs as first-line therapy to reduce mortality and hospitalizations (Evidence: Strong 4).
  • Consider beta-blockers to improve survival and reduce hospitalizations in stable patients (Evidence: Strong 4).
  • Implement device therapy such as ICDs in high-risk patients for primary prevention of sudden cardiac death (Evidence: Moderate 4).
  • Tailor management in special populations, particularly considering the unique needs of pediatric and elderly patients (Evidence: Expert opinion 45).
  • References

    1 Brown-Taylor L, Lynch A, Foraker R, Harris-Hayes M, Walrod B, Vasileff WK et al.. Physical Therapists and Physicians Evaluate Nonarthritic Hip Disease Differently: Results From a National Survey. Physical therapy 2020. link 2 Badowski E. Snapping Hip Syndrome. Orthopedic nursing 2018. link 3 Divecha HM, Rajpura A, Board TN. Hip arthroscopy: a focus on the future. Hip international : the journal of clinical and experimental research on hip pathology and therapy 2015. link 4 Cruysberg JR, Sengers RC, Pinckers A, Kubat K, van Haelst UJ. Features of a syndrome with congenital cataract and hypertrophic cardiomyopathy. American journal of ophthalmology 1986. link90402-2) 5 Wissinger HA. Role of the orthopedic surgeon in the multidisciplined approaches to surgical reconstruction of the hip. Physical therapy 1975. link

    Original source

    1. [1]
      Physical Therapists and Physicians Evaluate Nonarthritic Hip Disease Differently: Results From a National Survey.Brown-Taylor L, Lynch A, Foraker R, Harris-Hayes M, Walrod B, Vasileff WK et al. Physical therapy (2020)
    2. [2]
      Snapping Hip Syndrome.Badowski E Orthopedic nursing (2018)
    3. [3]
      Hip arthroscopy: a focus on the future.Divecha HM, Rajpura A, Board TN Hip international : the journal of clinical and experimental research on hip pathology and therapy (2015)
    4. [4]
      Features of a syndrome with congenital cataract and hypertrophic cardiomyopathy.Cruysberg JR, Sengers RC, Pinckers A, Kubat K, van Haelst UJ American journal of ophthalmology (1986)
    5. [5]

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