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Cardiac beriberi

Last edited: 4/14/2026

Overview

Cardiac beriberi, caused by severe thiamine (vitamin B1) deficiency, manifests primarily as wet beriberi with cardiac dysfunction, including high cardiac output, low systemic vascular resistance, and potential right-sided heart failure 1267. It can occur post-gastric surgery, in malnourished states, and among populations with restricted dietary intake or social isolation 14.

Diagnosis

  • Clinical Presentation: Symptoms include dyspnea, edema, palpitations, nausea, vomiting, and signs of heart failure 12467.
  • Hemodynamic Indices: High cardiac output and low systemic vascular resistance are indicative 267.
  • Laboratory Tests: Low serum thiamine levels are crucial for diagnosis 25.
  • Specific Conditions: Consider in patients with a history of malnutrition, gastric surgery, alcoholism, or social withdrawal 1467.
  • Differential Diagnosis: Rule out other causes of heart failure and elevated lactate 5.
  • Management

  • Thiamine Supplementation: Intravenous thiamine is first-line, with doses varying but often including bolus administration 267.
  • Supportive Care: Manage heart failure symptoms with standard therapies, adjusting based on hemodynamic response 267.
  • Dietary Intervention: Ensure adequate thiamine intake post-surgery or in malnourished states 1.
  • Monitoring: Regular assessment of thiamine levels and clinical improvement 5.
  • Special Populations

  • Post-Gastric Surgery Patients: Increased risk due to altered nutritional absorption; require close monitoring and supplementation 1.
  • Alcoholism: High risk for thiamine deficiency leading to severe forms like shoshin beriberi 67.
  • Social Isolation (Hikikomori Syndrome): Potential for thiamine deficiency due to restricted dietary intake 4.
  • Key Recommendations

  • Prompt Recognition and Thiamine Supplementation: Recognize subclinical thiamine deficiency, especially post-gastric surgery, and administer thiamine promptly to prevent morbidity and mortality (Evidence: Strong 1).
  • Consider Thiamine Deficiency in Non-Specific Cardiomyopathy: Include thiamine deficiency in the differential diagnosis for patients presenting with non-specific heart failure symptoms, particularly with hemodynamic instability (Evidence: Moderate 267).
  • Enhanced Nutritional Follow-Up: Implement rigorous nutritional follow-up protocols for patients post-gastric surgery to prevent beriberi (Evidence: Expert opinion 1).
  • References

    1 Wilson RB. Pathophysiology, prevention, and treatment of beriberi after gastric surgery. Nutrition reviews 2020. link 2 Lei Y, Zheng MH, Huang W, Zhang J, Lu Y. Wet beriberi with multiple organ failure remarkably reversed by thiamine administration: A case report and literature review. Medicine 2018. link 3 Lonsdale D. Thiamin. Advances in food and nutrition research 2018. link 4 Tanabe N, Hiraoka E, Kataoka J, Naito T, Matsumoto K, Arai J et al.. Wet Beriberi Associated with Hikikomori Syndrome. Journal of general internal medicine 2018. link 5 Duca J, Lum CJ, Lo AM. Elevated Lactate Secondary to Gastrointestinal Beriberi. Journal of general internal medicine 2016. link 6 Imamura T, Kinugawa K. Shoshin Beriberi With Low Cardiac Output and Hemodynamic Deterioration Treated Dramatically by Thiamine Administration. International heart journal 2015. link 7 Kim J, Park S, Kim JH, Kim SW, Kang WC, Kim SJ. A case of shoshin beriberi presenting as cardiogenic shock with diffuse ST-segment elevation, which dramatically improved after a single dose of thiamine. Cardiovascular journal of Africa 2014. link 8 Jukes TH. The prevention and conquest of scurvy, beri-beri, and pellagra. Preventive medicine 1989. link90023-6)

    Original source

    1. [1]
    2. [2]
    3. [3]
      Thiamin.Lonsdale D Advances in food and nutrition research (2018)
    4. [4]
      Wet Beriberi Associated with Hikikomori Syndrome.Tanabe N, Hiraoka E, Kataoka J, Naito T, Matsumoto K, Arai J et al. Journal of general internal medicine (2018)
    5. [5]
      Elevated Lactate Secondary to Gastrointestinal Beriberi.Duca J, Lum CJ, Lo AM Journal of general internal medicine (2016)
    6. [6]
    7. [7]
    8. [8]

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