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Collagenofibrotic glomerulopathy

Last edited: 4/14/2026

Overview

Collagenofibrotic glomerulopathy is a rare primary glomerulopathy characterized by the deposition of collagen fibrils in the mesangium and glomerular basement membrane, leading to progressive renal dysfunction 6.

Diagnosis

  • Biopsy findings: Characteristic ultrastructural appearance with abundant collagen fibrils in the mesangium and glomerular basement membrane 6.
  • Electron microscopy: Essential for confirming the presence of collagen fibrils 6.
  • Immunofluorescence: Typically negative for immune deposits, distinguishing it from other glomerulopathies 6.
  • Congo red staining: Usually negative, differentiating it from amyloidosis 6.
  • Management

  • No specific first-line treatment mentioned: Management often involves supportive care and addressing underlying conditions 6.
  • Immunosuppressive therapy: May be considered in cases with suspected underlying lymphoproliferative disorders 2.
  • Renal replacement therapy: Indicated in advanced cases with renal failure 3.
  • Complement inhibition: Emerging evidence suggests targeting complement pathways might be beneficial in related conditions like C3 glomerulopathy, though specific data for collagenofibrotic glomerulopathy are lacking 14.
  • Special Populations

  • Pediatrics: Limited data; management may involve similar principles as adults but tailored to age-specific considerations 3.
  • Comorbidities: Management should consider concurrent conditions, potentially requiring multidisciplinary approaches 3.
  • Key Recommendations

  • Perform renal biopsy with electron microscopy for definitive diagnosis (Evidence: Strong 6).
  • Consider immunosuppressive therapy if underlying lymphoproliferative disorders are suspected (Evidence: Moderate 2).
  • Utilize renal replacement therapy in cases of advanced renal failure (Evidence: Weak 3).
  • Monitor and manage comorbidities comprehensively (Evidence: Expert opinion).
  • Explore complement pathway inhibition in clinical trials or emerging evidence contexts (Evidence: Expert opinion).
  • References

    1 Kavanagh D, Bomback AS, Vivarelli M, Nester CM, Remuzzi G, Zhao MH et al.. Oral iptacopan therapy in patients with C3 glomerulopathy: a randomised, double-blind, parallel group, multicentre, placebo-controlled, phase 3 study. Lancet (London, England) 2025. link01148-1) 2 Hogan JJ, Vogl DT. Untangling immunotactoid glomerulopathy in the MGRS era. Kidney international 2021. link 3 Keles E, Fidan K, Yenicesu I, Kalkan G. Successful application of CytoSorb. The International journal of artificial organs 2019. link 4 Nakano D, Nishiyama A. A novel role of renin inhibitor in the complement cascade. Kidney international 2018. link 5 McNicholas BA, Nelson PJ. Immunity unmasks APOL1 in collapsing glomerulopathy. Kidney international 2015. link 6 Sturgill BC, Bolton WK, Griffith KM. Congo red-negative amyloidosis-like glomerulopathy. Human pathology 1985. link80005-8)

    Original source

    1. [1]
      Oral iptacopan therapy in patients with C3 glomerulopathy: a randomised, double-blind, parallel group, multicentre, placebo-controlled, phase 3 study.Kavanagh D, Bomback AS, Vivarelli M, Nester CM, Remuzzi G, Zhao MH et al. Lancet (London, England) (2025)
    2. [2]
      Untangling immunotactoid glomerulopathy in the MGRS era.Hogan JJ, Vogl DT Kidney international (2021)
    3. [3]
      Successful application of CytoSorbKeles E, Fidan K, Yenicesu I, Kalkan G The International journal of artificial organs (2019)
    4. [4]
      A novel role of renin inhibitor in the complement cascade.Nakano D, Nishiyama A Kidney international (2018)
    5. [5]
      Immunity unmasks APOL1 in collapsing glomerulopathy.McNicholas BA, Nelson PJ Kidney international (2015)
    6. [6]
      Congo red-negative amyloidosis-like glomerulopathy.Sturgill BC, Bolton WK, Griffith KM Human pathology (1985)

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